Autoimmune hazards of hepatitis B vaccine.

“According
to Hippocratic tradition, the safety level of a preventive medicine
must be very high, as it is aimed at protecting people against diseases
that they may not contract.” ~ Marc Girard, Autoimmune hazards of hepatitis B vaccine.



Startling
new research published in the journal Apoptosisindicates that
hepatitis B vaccine, which is designed to prevent Hepatitis B
virus-induced damage to the liver, actually causes liver cell
destruction.


In the study
titled “Hepatitis B vaccine induces apoptotic death in Hepa1-6
cells,” researchers set out to “...establish an in vitro model system
amenable to mechanistic investigations of cytotoxicity induced by
hepatitis B vaccine, and to investigate the mechanisms of
vaccine-induced cell death.”


They found the hepatitis
B vaccine induced a “loss of mitochondrial integrity, apoptosis
induction, and cell death” in liver cells exposed to a low dose of
adjuvanted hepatitis B vaccine. The adjuvant used was aluminum hydroxide, which is increasingly being identified as a contributing cause of autoimmune disease in immunized populations.


The discovery that the hepatitis B vaccine damages the liver (hepatotoxicity) confirms earlier findings
(1999) that the vaccine increases the incidence of liver problems in
U.S. children less than 6 years old by up to 294% versus unvaccinated
controls.
Another study
published in the journal Hepatogastroentology in 2002, observed that
Hepatitis B vaccination was statistically associated with
gastrointestinal reactions including: hepatitis, gastrointestinal disease and liver function test abnormalities in comparison to other vaccine control groups.


This, however, is only the tip of the iceberg...


In a revealing study published
in June 2011 in the journal Molecular Biology Reports, researchers
demonstrated that hepatitis B vaccine alters the expression of 144 genes
in the mouse liver within 1 day of vaccination, 7 of which are related
to inflammation and metabolism. The authors noted:



"Pharmaceutical
companies usually perform safety testing of vaccines, but all
requirements of the World Health Organization and drug pharmacopoeias
depend on general toxicity testing, and the gene expression study of
hepatitis B vaccine is not done routinely to test vaccine quality."




Could
the gene-expression altering affects of hepatitis B vaccine be one
reason why there are over 60 serious detrimental health effects
associated with the vaccine as documented in the peer-reviewed and published biomedical literature, including sudden infant death?



Other potential mechanisms of action behind hepatitis B vaccine's dangerous side effects, are as follows...

• Hepatitis
  B vaccines may contain Hepatitis B Virus polymerase as a contaminant,
  which may trigger an auto-immune process against the myelin
  (protective coating on the nerves) in some vaccinated subjects.
  SOURCES: PMID: 16176857, PMID: 15908138
  
• Hepatitis B vaccine may induced autoimmune
  demyelinating disease through the molecular mimicry that exists
  between the vaccine antigen, Epstein-Barr virus and human myelin.
  SOURCES: PMID: 17630224 PMID: 16295528
  

Why Are They Vaccinating Infants For Hepatitis B Virus?


The
real danger here is that universal vaccination against Hepatitis B
virus may be causing far more harm than good. It is actually our
youngest -- infants -- who are most at risk of being irreparably
harmed, as the CDC's vaccine schedule requires Hepatitis B vaccination
at birth, 1-2 months, and then again at 3-6 months of age.


Universal
hepatitis B vaccination was recommended for U.S. newborns in 1991,
despite contradictory safety findings. Perhaps not coincidentally, the
prevalence of autism today is 1500% higher than that occurring in the
period immediately before their introduction. While there is no such
thing as a "genetic epidemic," in the traditional inheritable sense of
the word "genetic," there is such a thing as environmentally induced
gene-expression changes, as described above. In other words, vaccine
adjuvants (e.g. mercury and aluminum) and vaccine antigens are
capable of profoundly affecting the stability of the genetic
infrastructure upon which our health depends.


According to one review
published in the Journal of Toxicology and Environmental Health in
2010, male newborns vaccinated with hepatitis B prior to 1999 had a
3-fold higher risk for parentally reported autism. Why before 1999? On
8/27/99 the CDC, in tacit acknowledgment of the profound neurotoxicity
associated with the use of thimerosal (organomercury), approved the
first thimerosal-free hepatitis B vaccine. Sadly, even after the removal
of mercury (which was replaced by another neurotoxic agent aluminum
hydroxide), autism prevalence is still several orders of magnitude
higher than it was before the CDC's increasingly overwhelming vaccine schedule (60+ by age 6) reached its present-day proportions.


Another
glaring problem with Hep. B vaccine in infants is that Hepatitis B
virus is only transmitted through blood or semen by those who are
infected, which are two routes of exposure an infant -- certainly not
one born in a hospital -- should ever be exposed to; unless, of course,
the mother is a carrier, and therefore can transmit it vertically to
her offspring. But hospitals can and should screen mothers for
Hepatitis B preemptively, therefore making it unnecessary to vaccinate
every infant blindly. In addition, there are no randomized controlled
trials that have assessed the effects of hepatitis B vaccine during
pregnancy for preventing infant infection, despite the fact that
pregnant women are being given the vaccine for exactly this reason.¹
There is also research indicating that immunization for Hepatitis B
does not guarantee protection against becoming infected with it; i.e.
it may not truly fall within the category of a vaccine-preventable
disease.


Source:-
http://www.greenmedinfo.com/blog/hep-b-vaccine-damages-liver-it-supposed-protect